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Alopecia areata is noncontagious or not contagious. It occurs with greater regularity in people who have affected family suggesting that heredity might be a cause. Strong evidence that genes may increase peril for alopecia areata was found by studying families with two or more affected members. This study identified at least 4 regions inside the genome which might be likely to contain alopecia areata genes. In addition it is somewhat more prone to occur in individuals who have relatives with auto immune diseases.

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The condition is assumed to be an autoimmune disorder when the natural object attacks its hair follicles and suppresses or stops new hair growth. There is evidence that T cell lymphocytes cluster around these follicles causing excitement and subsequent hair loss. An unknown environmental trigger for example emotional stress or possibly a pathogen is believed to combine with inherited genes to cause the trouble. At that place there are a few recorded cases of babies being born with congenital alopecia areata, but these are not cases of autoimmune disease because a child is born with no fully developed body's defence mechanism.

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